基因编辑权威指南。
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CRISPR basics

How cells repair DNA — and why it decides the edit

The cell, not the editor, makes the change. Which repair pathway it uses determines what you actually get.

简短解答

CRISPR breaks the DNA. What happens next is up to the cell, and it has several ways of fixing a break. The quick, messy one usually leaves a few letters wrong, which breaks the gene — useful if that is what you wanted. The careful one can copy in an exact replacement, but it only works in cells that are dividing, and even then it often does not. That is why 'fixing' a gene is so much harder than breaking one.

Double-strand break repair proceeds mainly by non-homologous end joining — active throughout the cell cycle, error-prone, producing indels — or homology-directed repair, restricted to S and G2 phase and requiring a homologous template. Microhomology-mediated end joining produces predictable deletions. Repair choice determines editing outcome, so it is manipulated experimentally by cell-cycle synchronisation, pathway inhibition and template design.

What can go wrong at a break

ImportantThese are the specific hazards that motivated base editing and prime editing, which nick rather than sever and so never trigger this repair cascade.

Sources

Connected in the Atlas

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Technologies

Prime Editing