The definitive guide to gene editing.
Menu
Home Learn News Ask the Atlas
Explore Technologies Diseases Treatments Clinical Trials Companies Scientists Genes Research Institutions
Beyond medicine Agriculture Ethics Investing World map
Learn & Tools Start here Glossary A–Z Compare technologies Timeline Lists & Rankings AI Agents ★ Saved API
About About us Methodology Data Sources Editorial Policy Contact Disclaimers

🧭 Guided View
New to genetics? We explain every term as you browse, in plain English. Same pages, with the help built in.

⚡ Expert View
You already know the biology. Just the content — clean and compact, no extra explanations. This is the default view.

Interface language
Light mode

Gene · Cardiovascular

LDLR

Low-density lipoprotein receptor — The receptor liver cells use to clear LDL cholesterol from the blood; faults in it are the commonest cause of familial hypercholesterolemia.

19p13.2 cholesterolin vivo
Chromosome 19 LDLR 19p13.2 p q

Schematic only — arm proportions are approximate and band detail is not drawn to scale. Cytogenetic location as published by NCBI Gene.

What this gene does

LDL cholesterol is removed from the blood by receptors on liver cells that catch it and pull it inside. LDLR is the gene for that receptor. If you inherit a faulty copy, you have fewer working receptors, cholesterol accumulates from childhood, and heart disease arrives decades early.

LDLR, at 19p13.2, encodes the low-density lipoprotein receptor mediating hepatic clearance of LDL particles. Loss-of-function variants are the most common cause of familial hypercholesterolemia; heterozygotes have roughly doubled LDL and markedly premature coronary disease. Editing strategies aim to increase LDLR activity — the mirror image of PCSK9 knockout, which achieves the same end indirectly.

Sources

  • MedlinePlus Genetics, U.S. National Library of Medicine
    LDLR gene ↗

Connected in the Atlas

Every entry on this site is linked to the others it relates to. These connections are part of the record, not a search result.