La guía definitiva sobre edición genética.
Menú
Inicio Aprender Noticias Pregunta al Atlas
Explorar Tecnologías Enfermedades Tratamientos Ensayos clínicos Empresas Científicos Genes Investigación Instituciones
Más allá de la medicina Agricultura Ética Inversión Mapa mundial
Aprender y herramientas Empieza aquí Glosario A–Z Comparar tecnologías Cronología Listas y rankings Agentes de IA ★ Guardado API
Acerca de Quiénes somos Metodología Fuentes de datos Política editorial Contacto Avisos legales

🧭 Vista guiada
¿Eres nuevo en genética? Explicamos cada término mientras navegas, en un lenguaje sencillo. Las mismas páginas, con la ayuda incorporada.

⚡ Opinión experta
Ya conoces la biología. Solo el contenido — limpio y compacto, sin explicaciones adicionales. Esta es la vista predeterminada.

Idioma de la interfaz
Modo claro

Technology · RNA targeting

CRISPR-Cas13

Targets RNA rather than DNA, so it can silence a gene's message without touching the genome itself.

Phase I RNAreversiblediagnostics
Clinical research Being tested in people in registered clinical trials. Being in trials is not evidence that a treatment works or is safe.

Explicación sencilla

Every other CRISPR system here goes after DNA — the permanent copy of the instructions. Cas13 goes after RNA, the working copies the cell makes from those instructions. Destroy the working copies and the protein stops being made, but the original is untouched. That means the effect wears off, which is sometimes exactly what you want: a treatment you can stop is safer than one you cannot.

Profundizar

Cas13 (type VI) effectors are RNA-guided RNA endonucleases with two HEPN domains. Guided by a crRNA to a complementary transcript, Cas13 cleaves it, knocking down gene expression without altering genomic DNA. Like Cas12a it shows collateral activity — against RNA — which is the basis of the SHERLOCK diagnostic platform. Catalytically dead Cas13 fusions enable programmable RNA base editing (REPAIR, RESCUE).

Why targeting RNA is different

A DNA edit is permanent by design: if it is wrong, it stays wrong. An RNA-targeting therapy has to be re-dosed, because the cell keeps making fresh transcripts — an inconvenience that doubles as a safety feature. It also means there is no risk of a heritable change and no double-strand break to repair.

The trade-off is that RNA targeting cannot fix anything. It reduces the amount of a protein being made. For conditions caused by a toxic protein that is useful; for conditions caused by a missing protein it is not.

Diagnostics

Cas13's collateral RNA cleavage powers SHERLOCK, which pairs isothermal amplification with a fluorescent reporter to detect specific nucleic-acid sequences at very low concentration — an approach deployed for viral detection including during the COVID-19 pandemic.

Diagnostics
A Cas13 effector engaging a single-stranded RNA transcript rather than DNA. Illustration generated for The CRISPR Atlas — a visual aid, not a photograph or a literal depiction of molecular structure.

Clinical status

Clinical work is early. Programmes have explored RNA knockdown in the eye and in genetic disease, and Cas13's reversibility makes it attractive where a permanent change would be hard to justify. Off-target and collateral activity in human cells has been a recurring concern in the literature and is an active area of engineering.

Sources

Connected in the Atlas

Every entry on this site is linked to the others it relates to. These connections are part of the record, not a search result.

Solo información educativa Esta página es una referencia, no asesoramiento médico. El estado de la investigación y la situación regulatoria cambian; comprueba la fecha de última actualización que aparece arriba y confirma cualquier dato importante con las fuentes primarias indicadas.